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  • FAK（局部粘着斑激酶）_百度百科
    局部粘着斑激酶（Focal Adhesion Kinase，FAK）是一种非受体型酪氨酸激酶，由FERM结构域、中央激酶区和C端FAT结构域组成，主要位于细胞粘着斑部位。 其编码基因位于8q24，具有Tyr397等6个可磷酸化酪氨酸位点，参与调控细胞粘附、迁移、增殖及凋亡等生理过程。
  • 黏着斑激酶 (FAK) - 知乎
    黏着斑激酶（FAK），也被称为PTK2蛋白酪氨酸激酶2（PTK2），由PTK2基因编码的蛋白质。 除某些类型的血细胞外，大多数细胞都表达FAK。 它活性引发细胞内信号转导途径，促进细胞接触与细胞外基质的转换，促进细胞迁移。 在发育过程中需要FAK，它的损失导致致死
  • Roles and inhibitors of FAK in cancer: current advances and future . . .
    Abstract Focal adhesion kinase (FAK) is a non-receptor tyrosine kinase that exhibits high expression in various tumors and is associated with a poor prognosis FAK activation promotes tumor growth, invasion, metastasis, and angiogenesis via both kinase-dependent and kinase-independent pathways
  • Recent advances in focal adhesion kinase (FAK)-targeting antitumor . . .
    FAK is widely expressed in human tissues and can regulate various cellular processes Overexpression of FAK can be detected in many different sources of human cancer cells Biological studies have shown that the activity of FAK is crucial for the occurrence and progression of human cancer
  • Targeting focal adhesion kinase (FAK) for cancer therapy: FAK . . .
    This review provides an overview of current drug discovery strategies targeting FAK, including the development of FAK inhibitors, FAK-based dual-target inhibitors and proteolysis-targeting chimeras (PROTACs) in both literature and patent applications
  • Discovery of potent focal adhesion kinase (FAK) inhibitor A8 with . . .
    FAK has emerged as a promising therapeutic target for cancer treatment due to its role in tumor survival, metastasis, and invasion Herein, we report the rational design, synthesis, and comprehensive evaluation of a novel FAK inhibitor, compound A8
  • FAK in cancer: mechanistic findings and clinical applications
    Focal adhesion kinase (FAK) is a cytoplasmic protein tyrosine kinase that is overexpressed and activated in several advanced-stage solid cancers
  • Design, Synthesis, Biological Evaluation and Molecular Docking . . . - MDPI
    Focal adhesion kinase (FAK) is often highly expressed across various types of tumors, making it a promising target for both therapy and diagnosis In this study, seven novel inhibitors were designed and synthesized





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