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  • Trialists Grapple with How to Outsmart TREM2 | ALZFORUM
    Series - AD PD™ 2025: Advances in Science Therapy: Part 1 of 15: Finally, PET Tracers For α-Synuclein Yield Signal in Idiopathic Parkinson’s Part 2 of 15: New Partnership Forms to Harness AI for Alzheimer’s Research Part 3 of 15: Moving Target: New Biology Casts TREM2 as a Shifty Mark Part 4 of 15: TREM2: A Chill Pill for Neurons? Part 5 of 15: Trialists Grapple with How to Outsmart
  • TREM2 H157Y - ALZFORUM
    The H157Y mutation reduced activation in response to phospholipid ligands in 2B4 T cells engineered to turn on GFP expression when TREM2 was engaged (Song et al , 2017) and also decreased phagocytosis of pHrodo-E Coli by HEK Flp-in cells expressing the variant (Schlepckow et al , 2017), compared with cells that expressed wild-type TREM2
  • TREM2 R47H - ALZFORUM
    Ghani M, Sato C, Kakhki EG, Gibbs JR, Traynor B, St George-Hyslop P, Rogaeva E Mutation analysis of the MS4A and TREM gene clusters in a case-control Alzheimer's disease data set
  • Two Paths for TREM2-Positive Microglia: DAM or Senescence?
    Senescent microglia in amyloidosis mice express high levels of TREM2 They have distinct protein profiles from disease-associated microglia (DAM) The two TREM2 subtypes are hard to distinguish in transcriptomic studies
  • All Roads Lead to TREM2: Gearing Up to Target This Receptor
    Since the discovery of rare, yet potent risk variants in the TREM2 gene 11 years ago, the microglial receptor has emerged as a pivot point in the pathogenesis of Alzheimer’s disease Findings presented at the International Conference on Alzheimer’s and Parkinson’s Diseases, held March 28 to
  • TREM2-mKATE2 Reporter Mouse | ALZFORUM
    Summary In this mouse, TREM2 and the fluorescent protein mKate2 are co-expressed under the control of the endogenous Trem2 promoter, allowing researchers to indirectly assess expression of TREM2 by measuring mKate2 fluorescence (Feiten et al , 2024)
  • Soluble TREM2 induces inflammatory responses and enhances . . . - ALZFORUM
    These findings, in light of the Zhong et al paper, illustrate that soluble TREM proteins do modulate inflammation and survival mechanisms Zhong and colleagues show that sTREM2 acts in an autocrine manner to drive the expression of NFκB-dependent proinflammatory cytokine expression and microglial survival
  • A candidate regulatory variant at the TREM gene cluster associates with . . .
    A candidate regulatory variant at the TREM gene cluster associates with decreased Alzheimer's disease risk and increased TREML1 and TREM2 brain gene expression
  • Trem2*R47H (NSS) - ALZFORUM
    Summary R47H is a rare variant in TREM2 that triples the risk of Alzheimer’s disease in heterozygous carriers In order to study the effects of this variant, several mouse models were created in which the R47H mutation was introduced into the mouse Trem2 gene (Trem2 R47H KI (JAX), Trem2 R47H KI (Haass), Trem2 R47H KI (Lamb Landreth)) Unfortunately, these early R47H knock-in mice had





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